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L'industrie pharmaceutique invente souvent des maladies pour creer un marche lucritif comme les fameuses maladies auto-immunes.
Plusieurs medecins respectables doutaient l'origine de ces maladies auto-immunes. Parmis ces medecins Dr Gustavo Bounous, doutait un deficit en glutathione et pas en corticostroides qui est responsable de ces maladies auto-immunes.
Dr Pottenger croit qu'il s'agit des infections endocellulaires qui infectent les tissues, et l'immunite en voulant detruire ces microbes endocellulaires detruit le tissu. On connait les cavernes de la tuberculoses dans les poumons.
La solution est simple. Il faut augmenter le glutathione cellulaire. Ici une preuve que le glutathione aide les cellules immunitaires cytotoxiques a detruire le virus de l'hepatite C et a reduire considerablement la viremie et ameliore tous les indices de l'inflammation.
Le glutathatione est consomme par les cellules cytotoxiques CD8, CD4, NK cells pour produire l'oxide de nitrogen. Une molecule capable de detruire tous les virus et sans risque de developpement de resistance.
Dr Gustavo Bounous avait montre dans des etudes a l'universite de McGill que la consommation de whey protein peut reduire la viremie du VIH chez les malades du SIDA considerablement.
Dr Kremer Heinrich en Allemagne avait les memes conclusions. Le glutathione cellulaire aide les cellules th1 a produire le NO pour detruire TOUS virus. C'est une mechanisme evolutionnaire stable et les virus sont incapables de devenir resistants au NO.
Plusieurs medecins respectables doutaient l'origine de ces maladies auto-immunes. Parmis ces medecins Dr Gustavo Bounous, doutait un deficit en glutathione et pas en corticostroides qui est responsable de ces maladies auto-immunes.
Dr Pottenger croit qu'il s'agit des infections endocellulaires qui infectent les tissues, et l'immunite en voulant detruire ces microbes endocellulaires detruit le tissu. On connait les cavernes de la tuberculoses dans les poumons.
La solution est simple. Il faut augmenter le glutathione cellulaire. Ici une preuve que le glutathione aide les cellules immunitaires cytotoxiques a detruire le virus de l'hepatite C et a reduire considerablement la viremie et ameliore tous les indices de l'inflammation.
Le glutathatione est consomme par les cellules cytotoxiques CD8, CD4, NK cells pour produire l'oxide de nitrogen. Une molecule capable de detruire tous les virus et sans risque de developpement de resistance.
Dr Gustavo Bounous avait montre dans des etudes a l'universite de McGill que la consommation de whey protein peut reduire la viremie du VIH chez les malades du SIDA considerablement.
Dr Kremer Heinrich en Allemagne avait les memes conclusions. Le glutathione cellulaire aide les cellules th1 a produire le NO pour detruire TOUS virus. C'est une mechanisme evolutionnaire stable et les virus sont incapables de devenir resistants au NO.
The use of whey protein concentrate in management of chronic hepatitis C virus – a pilot study
Gamal Elattar,corresponding author1 Zeinab Saleh,2 Safinaz EL-Shebini,2 Atif Farrag,2 Mona Zoheiry,1 Azza Hassanein,1 Maged EL-Ghannam,1 Shendy Shendy,1 Ehab EL-Dabaa,1 and Nariman Zahran1
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Abstract
Introduction
Whey protein contains biologically active ingredients that can prevent and attenuate disease besides being nutritive. The aim of the study was to clarify the effects of oral administration of whey protein on viral load and host defence mechanisms, in particular, phagocytic function of neutrophils, selected immunomodulatory cytokines and serum inflammatory markers, in compensated chronic hepatitis C virus (HCV) patients.
Material and methods
Twenty-seven HCV patients (20 males and 7 females) recruited from the hepatology clinic of the Theodor Bilharz Research Institute (TBRI) were given whey protein concentrate (WPC) twice daily for two months. In addition, 15 age and sex matched healthy participants were included in the study, as a control group. Neutrophil phagocytic activity, serum intercellular adhesion molecule (sICAM), interleukin-2 (IL-2), nitric oxide (NO), as well as HCV-RNA levels and routine investigations were determined for patients, before and after WPC supplementation and once for the control group.
Results
There was a significant decrease in viral load and markers of active inflammation, alanine aminotransferase (ALT) and aspartate aminotransferase (AST), while serum albumin, total leucocyte counts and absolute neutrophil counts showed significant elevation accompanied by improvement of neutrophil phagocytic activity after WPC supplementation compared to pre-treated levels. The oral WPC supplementation was well tolerated without any serious adverse events.
Conclusions
Oral supplementation of WPC has promising results as a new therapeutic strategy against HCV and its sequelae by decreasing the viral load and active inflammation as well as improving the synthetic capacity of the liver and the phagocytic function of neutrophils, in these patients.
Gamal Elattar,corresponding author1 Zeinab Saleh,2 Safinaz EL-Shebini,2 Atif Farrag,2 Mona Zoheiry,1 Azza Hassanein,1 Maged EL-Ghannam,1 Shendy Shendy,1 Ehab EL-Dabaa,1 and Nariman Zahran1
Author information ► Article notes ► Copyright and License information ► Disclaimer
This article has been cited by other articles in PMC.
Go to:
Abstract
Introduction
Whey protein contains biologically active ingredients that can prevent and attenuate disease besides being nutritive. The aim of the study was to clarify the effects of oral administration of whey protein on viral load and host defence mechanisms, in particular, phagocytic function of neutrophils, selected immunomodulatory cytokines and serum inflammatory markers, in compensated chronic hepatitis C virus (HCV) patients.
Material and methods
Twenty-seven HCV patients (20 males and 7 females) recruited from the hepatology clinic of the Theodor Bilharz Research Institute (TBRI) were given whey protein concentrate (WPC) twice daily for two months. In addition, 15 age and sex matched healthy participants were included in the study, as a control group. Neutrophil phagocytic activity, serum intercellular adhesion molecule (sICAM), interleukin-2 (IL-2), nitric oxide (NO), as well as HCV-RNA levels and routine investigations were determined for patients, before and after WPC supplementation and once for the control group.
Results
There was a significant decrease in viral load and markers of active inflammation, alanine aminotransferase (ALT) and aspartate aminotransferase (AST), while serum albumin, total leucocyte counts and absolute neutrophil counts showed significant elevation accompanied by improvement of neutrophil phagocytic activity after WPC supplementation compared to pre-treated levels. The oral WPC supplementation was well tolerated without any serious adverse events.
Conclusions
Oral supplementation of WPC has promising results as a new therapeutic strategy against HCV and its sequelae by decreasing the viral load and active inflammation as well as improving the synthetic capacity of the liver and the phagocytic function of neutrophils, in these patients.
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